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Cellular & Molecular Neurobiology
One editor, K journal formats. With the largest collection of verified journal formats, what you need is already there. I spent hours with MS word for reformatting. It was frustrating - plain and simple. Joseph M. Katz Graduate School of Business. School of Computing and Information. School of Dental Medicine. School of Education. Swanson School of Engineering. School of Health and Rehabilitation Sciences. School of Information Sciences. School of Law. School of Medicine.
School of Nursing. School of Pharmacy. Graduate School of Public and International Affairs. Graduate School of Public Health. School of Social Work. University Center for International Studies. Investigating the cellular and molecular mechanisms that control the proliferation and differentiation of neural precursors in the developing and mature retina.
Aims and Scope
Our research goals are to understand how ion channels are precisely localized to control neuronal excitability and how localization and function of ion channels are altered in neurodegenerative diseases. Gene therapy for dominant genetic diseases using RNA interference RNAi , with particular focus on muscular dystrophy and neurodegenerative disease.
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Role of cell adhesion molecules in the processes of synaptogenesis and circuit assembly in the developing zebrafish nervous system. We are studying how a circadian hour clock modulates cellular and molecular processes and chemical and electrical synaptic transmission, and how disruption of this circadian system mediates neuronal degeneration. We are also studying the cellular, subcellular eg transporters , developmental, and neural network mechanisms that underlie information processing in the brain. Research in the Martin lab is focused on the role of glycoyslation in synapse formation and muscular dystrophy.
Other studies involve understanding the role of carbohydrates in the development of the brain, and the development of diagnostic and therapeutic reagents for Alzheimer's disease. My research aims to better understand and treat the maladaptive immune response after spinal cord injury. This is composed of i the systemic spinal cord injury-induced immune deficiency syndrome SCI-IDS , ii consecutive infections and ii the developing post-traumatic autoimmunity.
Both maladaptive neuro-immunological syndromes and their consequences are contributing to the underlying neuropathology and represent a candidate target to improve neurological recovery. MicroRNA biology, tissue injury and repair, regenerative medicine, nutrition, oxygen and hypoxia, wound healing, stroke and neurodegeneration, myocardial infarction.